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Extra resources for Kaplan USMLE Step 2: Infectious Diseases

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The report from the Quality Standards Subcommittee (Knopman et al. 2001), reviewing procedures for the evaluation of dementia, supported both the DSM and National Institute of Neurologic and Communicative Disorders and Stroke—Alzheimer’s Disease and Related Disorders Association criteria as reliable and valid for the diagnosis of Alzheimer dementia (guideline). , in the primary care setting—Patterson et al. 2001, see chap. 3—versus the specialty setting—Knopman et al. 2001). Cognitive and neuroanatomic changes occur as part of the aging process.

Schaie. 1990. Handbook of the Psychology of Aging, 3rd ed. San Diego: Academic Press. , A. V. Williams. 1980. Behavioral slowing with age: Causes, organization, and consequences. In Aging in the 1980s, edited by L. Poon. : American Psychological Association, pp. 293–308. E. Tomlinson, and M. Roth. 1968. The associations between quantitative measures of dementia and of senile change in the cerebral grey matter of elderly subjects. British Journal of Psychiatry 114:797–811. J. de Leon, A. Convit, et al.

Regionspeci~c studies show greater neuronal decreases in some areas, including the superior frontal and temporal gyrus, precentral gyrus, visual cortex, locus ceruleus, substantia nigra, basal nucleus of Meynert, and cerebellar Purkinje cells (Lauter 1985). In primary degenerative dementia, the decrease in brain weight is as much as 10% more than that seen in normal aging (Terry and Davies 1983). As in normal aging, atrophy and eventual cell loss is region-speci~c in DAT. Brains from persons with DAT demonstrate greater neuronal loss in frontal and temporal regions (Mountjoy et al.

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