Download e-book for iPad: Medical otology and neurotology: a clinical guide to by Mohamed Hamid, Aristides Sismanis
By Mohamed Hamid, Aristides Sismanis
Finished overview of the remedy of internal and heart ear disorder and the ensuing listening to and stability problems. Covers the scientific functions of state of the art learn, together with regenerative drugs for internal ear affliction, internal ear perfusion remedies, vestibular rehabilitation for stability retraining, and using cochlear impants. Covers prognosis and administration of peripheral auditory and vestibular issues, relevant vestibular issues, problems of the facial nerve, and more. Read more...
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Additional resources for Medical otology and neurotology: a clinical guide to auditory and vestibular disorders
It also is known that these peptides are vasoactive and inﬂuence the integrity of the blood–brain barrier, making the stria blood–labyrinth barrier a likely site of inﬂuence as well. Certainly other ion channels are at work in the inner ear and will be characterized by future research, as will the hormonal and genetic factors that control them. Nevertheless, current clinical and experimental otology research is helping to deﬁne the role of these channels and how their function is affected by various disease processes.
Genes are listed with their respective loci. Gene products, if known, are in parentheses (Adapted from Van Laer L, Cryns K, Smith RJH, Van Camp G, Nonsyndromic hearing loss. Ear Hear 2003;24:275–288 and Van Camp G, Smith RJH. Hereditary Hearing Loss homepage. be/dnalab/hhh/). 3,9 The ﬁbrocytes of the lateral wall and spiral ligament move K+ through gap junctions that are made up of various connexin proteins (Fig. 3–4). Multiple genes and loci have now been identiﬁed as responsible for at least ﬁve different gap junction connexin proteins.
Genet Med 2002;4:258–274 28. Denoyelle F, Marlin S, Weil D, et al. Clinical features of the prevalent form of childhood deafness, DFNB1, due to a connexin-26 gene defect: implications for genetic counselling. Lancet 1999;353:1298–1303 29. Cohn ES, Kelley PM, Fowler TW, et al. Clinical studies of families with hearing loss attributable to mutations in the connexin 26 gene (GJB2/DFNB1). Pediatrics 1999;103: 546–550 30. Feldmann D, Denoyelle F, Loundon N, et al. Clinical evidence of the nonpathogenic nature of the M34T variant in the connexin 26 gene.